I totally understand what kind of job you had. I work at a Wally World distribution center. Started in shipping, loading about 3 to 4 full semi’s a day. Didn’t know how much weight I loaded. Then switched to non conveyable. When in dog food, I’d stack about 60k/lbs in 12 hours. That was MUCH easier than shipping. My first year, I struggled. Then I talked to my bro-in-law, who is a personal trainer, found and I started to do good. I was taking creatine and C4 prior to work, and took an Animal Pak with UniLiver every break, while eating protien every 3-4 hours. This brought me to Muscle for Life and The Books. I’m in maintenance department now, and are about to join a gym. I’ve been wanting to get the Legion multi’s and switch to Legion supplements. I’m about done reading BLS, and are gonna start the year one challenge. I’ve aready bought BBLS and Shredded Chef. I get excited every time I think about my goals.
Anabolic-androgenic steroid (AAS) withdrawal is established to be an important, though poorly known medical problem, because of AAS potency to cause physical and psychological dependence. Thus discontinuation of high-dose, long-term anabolic steroid use, apart from endocrine dysfunction (hypogonadotropic hypogonadism), may lead to development of withdrawal symptoms. They include mood disorders (with suicidal depression as the most life-threatening complication), insomnia, anorexia, decreased libido, fatigue, headache, muscle and joint pain, and desire to take more steroids. The withdrawal from anabolic steroids usually requires treatment. Clinical management, as with other drugs of abuse, consists of supportive therapy and pharmacotherapy. The goals of treatment are to restore endocrine (hypothalamic-pituitary-gonadal, HPG) function and to alleviate withdrawal symptoms. The endocrine medications that are targeted specifically to ameliorate HPG function include testosterone esters, human chorionic gonadotropin, synthetic analogues of gonadotropin-releasing hormone and antiestrogens. They are indicated in the presence of persistent clinical symptoms or/and laboratory evidence of HPG dysfunction. Other medications, that are targeted to provide symptomatic relief include antidepressants (especially serotonin selective re-uptake inhibitors), nonsteroidal anti-inflammatory drugs and clonidine. Notwithstanding, it should be remembered that many of the above mentioned drugs have their own potential for abuse or side-effects, so their use must be carefully weighted and optimal treatment strategies for AAS withdrawal must await further clinical research.